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Senior Investigator

Yogita Chudasama, Ph.D.

Section on Behavioral Neuroscience


Building 35A Room 2D-820
35 Convent Drive
MD 20892-3749
Office: (301) 594-4186

Fax: (301) 480-8348
yogita.chudasama@nih.gov

Dr. Chudasama received her B.Sc (Hons) in Applied Psychology in 1991 and her Ph.D in 1995 from Cardiff University, Wales, UK.  Her Ph.D. was under the mentorship of Dr. Janice Muir where she studied prefrontal and thalamic contributions to learning, memory and attention. Her first postdoctoral fellowship (1998-2002) was in the Department of Experimental Psychology, University of Cambridge UK, with Prof. Trevor Robbins investigating psychopharmacological mechanisms of cognitive function. Her second postdoctoral fellowship (2003-2006) was in the Laboratory of Neuropsychology at NIMH with Dr. Elisabeth Murray examining the emotional and cognitive contributions of the prefrontal cortex and temporal lobe. In 2007, she joined the faculty at McGill University, Department of Psychology in Montreal, Canada. In November 2015, Dr. Chudasama moved to NIMH to establish the Section on Behavioral Neuroscience, and the NIMH Rodent Behavioral Core.  Her research is dedicated towards understanding the neural circuitry underlying the control of complex cognitive and emotional behavior.



Dr. Chudasama has had a long standing interest in functional neuroanatomy, and specifically elucidating the brain mechanisms underlying the control of executive function, which are both central to human cognitive behavior and adversely affected in psychiatric disorders. Her laboratory focuses on the cognitive effects of manipulating particular brain circuits, and is at the forefront in state-of-the-art behavioral testing. While her laboratory pursues basic science questions, Dr. Chudasama’s work places a strong emphasis on translation to understanding human brain circuits, cognition, and dysfunction observed in mental disorders. Recently, work in her work has extended towards understanding the relationship between executive dysfunction and socioemotional dysregulation. Currently, her lab is actively investigating the network of forebrain and midbrain structures that not only steer complex executive behavior, but also regulate normal social development. To this end, Dr. Chudasama’s laboratory implements a range of methodologies including behavior, neuroanatomy and psychopharmacology, as well as viral and molecular genetics to provide us with a unique perspective on the detailed circuits that support executive behaviors that often go awry in human patients suffering from poor control over their decisions, memories, and actions.

Staff Image
  • Kevin Cravedi, M.Sc
    Research Assistant

  • Johann du Hoffmann, Ph.D.
    Staff Scientist

  • Gloria Laryea, Ph.D.
    Postdoctoral Fellow

  • Michael Leventhal, B.S.
    Post baccalaureate IRTA Fellow

  • Fany Messanvi, Ph.D.
    Postdoctoral Fellow

  • Madeleine Perkins, B.S.
    Post baccalaureate IRTA Fellow

  • 1) Wilson EN, Abela AR, Do Carmo S, Allard S, Marks AR, Welikovitch LA, Ducatenzeiler A, Chudasama Y, Cuello AC. (2016)
  • Intraneuronal Aβ accumulation disrupts Hippocampal CRTC1-dependent gene expression and cognitive function in a rat model of Alzheimer’s disease.
  • Cerebral Cortex, In press.
  • 2) Prasad JA, Abela AR, Chudasama Y. (2016)
  • Midline thalamic reuniens lesions improve executive behavior.
  • Neuroscience, In press.
  • 3) Abela AR, Duan Y, Chudasama Y. (2015)
  • Hippocampal interplay with nucleus accumbens is critical for decisions about time.
  • European Journal of Neuroscience, 42, 2224-2233.
  • 4) Jang A, Costa V, Rudebeck P, Chudasama Y, Murray EA, Averbeck B. (2015)
  • Role of frontal cortical and medial-temporal lobe brain areas in learning a Bayesian prior belief on reversals.
  • Journal of Neuroscience, 35, 11751-11760.
  • 5) Chudasama Y. (2015)
  • Short-term and working memory in animals.
  • In: Ian P. Stolerman and Lawrence H. Price (2nd ed). Encyclopedia of Psychopharmacology, 1586, Springer-Verlag: Berlin Heidelberg.
  • 6) Abela AR, Chudasama Y. (2014)
  • Noradrenergic alpha-2a-receptor stimulation of the ventral hippocampus modulates impulsive decision-making.
  • Psychopharmacology, 231, 521-531.
  • 7) Mitchell AS. Sherman SM, Sommer MA, Mair RG, Vertes RP, Chudasama Y. (2014)
  • Advances in understanding mechanisms of thalamic relays in cognition and behavior.
  • Journal of Neuroscience, 34, 15340-15346.
  • 8) Prasad JA, Macgregor EM, Chudasama Y. (2013)
  • Lesions of the thalamic reuniens cause impulsive but not compulsive responses.
  • Brain, Structure and Function, 218, 85-96.
  • 9) Abela AR, Chudasama Y. (2013)
  • Dissociable contributions of the ventral hippocampus and orbitofrontal cortex to decision-making with a delayed or uncertain outcome.
  • European Journal of Neuroscience, 37, 360-647.
  • 10) Abela AR, Dougherty SD, Fagen ED, Hill CJR, Chudasama Y. (2013)
  • Inhibitory control deficits in rats with ventral hippocampal lesions.
  • Cerebral Cortex, 23, 1396-1409.
  • 11) Prasad JA, Chudasama Y. (2013)
  • Viral tracing identifies parallel disynaptic pathways to the hippocampus.
  • Journal of Neuroscience, 33, 8494-8503.
  • 12) Chudasama Y, Doobay, VM, Liu Y. (2012)
  • Hippocampal-prefrontal cortical circuit mediates inhibitory response control in the rat.
  • Journal of Neuroscience , 32, 10915-10924.
  • 13) Chudasama Y. (2011)
  • Animal models of prefrontal-executive function.
  • Behavioral Neuroscience, 125, 327-343.
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